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(R)-noradrenaline51-41-2 CAS NO.51-41-2
- FOB Price: USD: 85.00-90.00 /Kilogram Get Latest Price
- Min.Order: 1 Kilogram
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- Available Specifications:
98%(1-10)Kilogram98%(11-30)Kilogram
- Product Details
Keywords
- 51-41-2
- (R)-noradrenaline
- DL-norepinephrine
Quick Details
- ProName: (R)-noradrenaline51-41-2
- CasNo: 51-41-2
- Molecular Formula: C8H11NO3
- Appearance: crystallization
- Application: An antishock vasoactive drug. Mainly u...
- DeliveryTime: Within 3 working days after receiving...
- PackAge: 1kg/one bag, or 5kg/one bag,innermost ...
- Port: China main port
- ProductionCapacity: 10000 Metric Ton/Year
- Purity: 98%
- Storage: 2-8°C
- Transportation: Air, sea
- LimitNum: 1 Kilogram
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Details
Introduction:
English name:(R)-noradrenaline
CAS number: 51-41-2
Purity: 98 %
Appearance: Crystallization
Purpose: Angioshock-resistant vascular active drug. Mainly used to rescue acute hypotension and shock caused by peripheral vasodilation, etc..
Storage conditions: 2-8º C
CAS number: 51-41-2
Purity: 98 %
Appearance: Crystallization
Purpose: Angioshock-resistant vascular active drug. Mainly used to rescue acute hypotension and shock caused by peripheral vasodilation, etc..
Storage conditions: 2-8º C
Pharmacological effects
This product is a strong alpha receptor stimulant, which has a weak effect on the β1 receptor and has little effect on the β2 receptor. Through the excitation of alpha receptors, small arteries and small veins can be caused by vasoconstriction. The degree of vasoconstriction is related to alpha receptors on blood vessels. The skin mucosa vasoconstriction is the most obvious, followed by renal blood vessels, and the effect on coronary arteries is not obvious. This may increase with cardiac metabolites. The expansion of the coronary artery is related to the effect of this product. Through the excitement of the β1 receptor, the myocardial contraction was strengthened and the heart rate increased, but the effect was much weaker than that of epinephrine. The range of vasoconstriction caused by alpha receptor excitation is very wide, with skin, mucosal blood vessels, and glomeruli as the most obvious, followed by brain, liver, enteric membrane, skeletal muscle, etc.. After cardiac excitement, the cardiac metabolite adenosine is increased. Adenosine can promote coronary artery expansion. The cardiac aspect of alpha receptor excitation is mainly the enhancement of myocardial systolic force, rapid heart rate, and increased cardiac blood output; In the overall case, due to high elevated pressure, reflex excited vagus nerves can be caused to slow the heart rate and reduce the heart rate. The application of atropine can prevent this heart rate from slowing down. Because of the strong contraction of blood vessels, the peripheral resistance increases, so the output of the heart remains unchanged or decreases. Large doses can also cause arrhythmia, but are rare. Blood pressure: peripheral vasoconstriction and increased myocardial contractility cause increased blood supply, resulting in increased systolic and diastolic pressure, and slightly increased pulse pressure. Other: The effect on other smooth muscles is weak, but it can increase the frequency of uterine contraction in pregnant women, and the effect on body metabolism is also weak. Blood sugar increases only at large doses. Due to the difficulty of passing through the blood-brain barrier, there is little central effect. Excessive or lasting use can make capillary contraction, leakage of body fluids and reduced hemolysis.
This product is a strong alpha receptor stimulant, which has a weak effect on the β1 receptor and has little effect on the β2 receptor. Through the excitation of alpha receptors, small arteries and small veins can be caused by vasoconstriction. The degree of vasoconstriction is related to alpha receptors on blood vessels. The skin mucosa vasoconstriction is the most obvious, followed by renal blood vessels, and the effect on coronary arteries is not obvious. This may increase with cardiac metabolites. The expansion of the coronary artery is related to the effect of this product. Through the excitement of the β1 receptor, the myocardial contraction was strengthened and the heart rate increased, but the effect was much weaker than that of epinephrine. The range of vasoconstriction caused by alpha receptor excitation is very wide, with skin, mucosal blood vessels, and glomeruli as the most obvious, followed by brain, liver, enteric membrane, skeletal muscle, etc.. After cardiac excitement, the cardiac metabolite adenosine is increased. Adenosine can promote coronary artery expansion. The cardiac aspect of alpha receptor excitation is mainly the enhancement of myocardial systolic force, rapid heart rate, and increased cardiac blood output; In the overall case, due to high elevated pressure, reflex excited vagus nerves can be caused to slow the heart rate and reduce the heart rate. The application of atropine can prevent this heart rate from slowing down. Because of the strong contraction of blood vessels, the peripheral resistance increases, so the output of the heart remains unchanged or decreases. Large doses can also cause arrhythmia, but are rare. Blood pressure: peripheral vasoconstriction and increased myocardial contractility cause increased blood supply, resulting in increased systolic and diastolic pressure, and slightly increased pulse pressure. Other: The effect on other smooth muscles is weak, but it can increase the frequency of uterine contraction in pregnant women, and the effect on body metabolism is also weak. Blood sugar increases only at large doses. Due to the difficulty of passing through the blood-brain barrier, there is little central effect. Excessive or lasting use can make capillary contraction, leakage of body fluids and reduced hemolysis.
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